All outcomes
Learning

Pathology Mastery

12 weeks · 0 milestones

Demonstrate understanding of disease mechanisms and histopathological features. Proof requires: (a) a histology identification log — minimum 30 slides identified with pathological features described, using a recognised digital pathology resource (PathPresenter, PathologyOutlines, or equivalent) with slide IDs logged for verification; and (b) a Q-bank session log showing documented performance of ≥70% on at least 50 pathology MCQs from a recognised Q-bank (Amboss, Pathoma, or equivalent), with screenshots of the session results showing date, question count, and score. The 70% threshold reflects the safety-critical context of pathology knowledge. Peer review is acceptable for this outcome as the verification systems (digital pathology platforms, Q-bank sessions) provide independent external confirmation of performance.

Milestone map

Milestone map

3 milestones

Produce a comprehensive knowledge map of general and systemic pathology covering the core mechanisms: cell injury and death (reversible injury, necrosis, apoptosis — types and triggers), inflammation (acute and chronic, cellular mediators, resolution vs. fibrosis), neoplasia (benign vs. malignant, hallmarks of cancer, tumour staging TNM), haemodynamic disorders (thrombosis, embolism, infarction, oedema), and immune-mediated disease (Type I–IV hypersensitivity with examples of each). For each mechanism, map: definition, cellular/molecular mechanism, clinical consequence, and a canonical clinical example.

Proof required

Submit your pathology knowledge map (table, concept map, or annotated diagram) covering all five mechanisms. Present to a qualified examiner who tests recall of any mechanism and its clinical consequence.

What gets checked

  • All five mechanism categories are covered — missing haemodynamic disorders or immune-mediated disease is a common shortcut.
  • Cellular mechanism is specified — 'causes tissue damage' is not a mechanism; 'neutrophil-mediated oxidative burst with reactive oxygen species production' is.
  • Clinical examples are specific — 'causes cancer' is not a clinical example; 'squamous cell carcinoma arising from chronic squamous metaplasia in smokers' is.

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